By Pierre Alain Gevenois, Paul de Vuyst
The spectrum of occupational and environmental illnesses has replaced markedly lately. New commercial procedures have ended in the construction and use of a variety of chemical compounds, metals, and alloys, more and more that have been pronounced to reason interstitial lung disorder in uncovered employees. hence, whereas the team in coal mining and asbestos dealing with has diminished, new teams of staff are prone to publicity to brokers very likely answerable for pneumoconiosis. This well-illustrated publication, written via the world over acclaimed specialists, presents a accomplished method of glossy imaging of environmental and occupational illnesses of the chest. the 1st a part of the publication addresses the elemental wisdom required to appreciate imaging during this context, whereas the second one makes a speciality of the imaging effects accomplished in various particular issues. there's specific emphasis on thin-section computed tomography considering that this method allows the detection of early subclinical abnormalities.
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Additional resources for Imaging of Occupational and Environmental Disorders of the Chest (Medical Radiology Diagnostic Imaging)
Example text
Although advanced disease often bears some microscopic resemblance to usual interstitial pneumonia, fibroblast foci are rare in asbestosis. As well, some cases show a more even distribution of fibrosis that mimics nonspecific interstitial pneumonia. It has been suggested (Craighead et al. 1982) that one should observe at least two asbestos bodies in tissue sections before rendering a diagnosis of asbestosis to avoid overdiagnosing asbestosis because of asbestos bodies created by background asbestos exposure to the general population.
The presence of asbestosis allows one to attribute the lung cancer to asbestos exposure. From Churg and Green (1998), used with permission Pathological Reactions to Inhaled Particles and Fibers chiole to bronchiole; however, in my opinion, fibrosis of the walls of the respiratory bronchioles and alveolar ducts represents a nonspecific reaction to many mineral dusts and is not asbestosis (reviewed in Churg and Wright 2003). Thus, true interstitial fibrosis must be seen to allow a diagnosis of asbestosis.
Considerable effort has been devoted in the literature to the proposition that asbestos-associated lung 26 cancers are of a particular histological cell type or have a particular location in the lung. Unfortunately, careful examination of the literature shows that there are no consistent differences in tumor location between tumors arising in workers with asbestos exposure compared with ordinary cigarette smokers (Kannerstein and Churg 1972; Whitwell et al. 1974; Karjalainen et al. 1993; Auerbach et al.