By Roy C. Orlando
This distinctive monograph encompasses the epidemiology, pathogenesis, analysis, administration, and issues of gastroesophageal reflux disorder (GERD). With insurance unprecedented in other places, Gastroesophageal Reflux disorder concentrates on a number of issues sharing the typical pathogenesis of gastroesophageal reflux, together with issues of the esophagus, oropharynx, larynx, and top respiration tract. This imperative reference ·compares GERD's pathogenesis from conventional (motor) and atraditional (epithelial) ways ·profiles uneven stipulations in young children and adults ·highlights precursors of esophageal adenocarcinoma ·explains the mechanisms of ways acid damages the esophageal epithelium ·illustrates GERD's courting to extra-esophageal stipulations resembling laryngitis, power bronchitis, and bronchial asthma ·and extra! Written via the world over famous experts and supplemented with over 1200 literature references, drawings, photos, X-rays, and tables, Gastroesophageal Reflux disorder is an important addition to the library of gastroenterologists, internists, normal and thoracic surgeons, kin practitioners, pediatricians, surgical and clinical citizens, pharmacists and pharmacologists, pulmonologists, otolarynogologists, oncologists, pathologists, and graduate and scientific institution scholars in those disciplines.
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Extra info for Gastroesophageal Reflux Disease (Gastroenterology and Hepatology)
Heartburn and regurgitation were found to be more common in the group with hernias larger than 5 cm. Patients with hernias greater than 3 cm had shorter LES with lower resting pressure and lower contraction amplitudes in the distal esophagus than patients with no hernia or hernias smaller than 3 cm. The patients with longer hernias also had more esophageal acid exposure and reduced acid clearance in comparison to those with small or no hernias. Ott et al. have also published a large series of 319 patients who had undergone barium esophagogram and 24-h pH monitoring to determine the correlation between presence and size of hiatal hernia using 24-h pH monitoring as a measure of degree of gastroesophageal reﬂux (15).
The authors concluded that recommending cessation of smoking was reasonable but questioned whether improvement in overall esophagitis would be noted. Risk Factors for GERD 43 In another study by Kahrilas and Gupta (52), esophageal manometry and pH monitoring were performed to determine the effect of smoking on LES function in eight normal nonsmokers, nine asymptomatic smokers, and nine smokers with hiatal hernia and endoscopic or histological evidence of esophagitis. Baseline manometry revealed that LES pressures were lowest in the smokers with GERD, higher in asymptomatic smokers, and highest in the nonsmoking controls.
Decktor DL, Allen ML, Robinson M. Esophageal motility, heartburn, and gastroesophageal reﬂux: variations in clinical presentation of esophageal dysphagia. Dysphagia 1990; 5:211–215. 46. Singh S, Stein HJ, DeMeester TR, Hinder RA. Nonobstructive dysphagia in gastroesophageal reﬂux disease: a study with combined ambulatory pH and motility monitoring. Am J Gastroenterol 1992; 87:562–567. 47. Jacob P, Kahrilas PJ, Vanagunas A. Peristaltic dysfunction associated with nonobstructive dysphagia in reﬂux disease.